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That notwithstanding, both subtypes of MBL can carry ‘CLL-specific’ genomic aberrations for example cytogenetic abnormalities and gene mutations, still to some much lesser extent when compared with CLL. These results recommend that this sort of aberrations are mostly applicable for disorder progression instead of condition onset, indirectly pointing to microenvironmental push like a vital contributor towards the emergence of MBL. Understanding microenvironmental interactions is as a result expected to elucidate MBL ontogeny and, most importantly, the relationship involving MBL and CLL.
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mutations MBL77 and complex kar yotype. It follows a linear evolution in the CLL clone through the recurrent acquisition of CDKN2A